Objective To investigate the natural susceptibility to 69 antimicrobial agents of 107 Enterobacter strains comprising E. amnigenus (n = 18), E. cancerogenus (n = 26), E. gergoviae (n = 28) and E. sakazakii (n = 35).

In this study, 77 Enterobacter spp.



This led to the absence of strains of intermediate susceptibility to tetracyclines in the presence of CAMHB. When the natural population was sensitive or intermediate according to the cited standard, it was described as naturally sensitive or naturally intermediate, respectively.






The resistance can vary from moderate to severe. Considerations for empirical therapy include an assessment regarding potential resistance to antibiotics, the infection site, anticipated achievable tissue concentrations of antibiotic, and predicted antibiotic adverse effects.

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Examples of germs in the Enterobacteriaceae family include Escherichia coli (E. coli) and Klebsiella pneumoniae..



clinical isolates from eight hospitals in Korea. This identification system contains biochemical key reactions for most clinically significant Enterobacteriaceae species. Although there is some information on antimicrobial susceptibility patterns of recently established The aim of the present study was to establish a database concerning the natural susceptibility to a wide range of antibiotics of strains of The identification of the strains was confirmed with a commercial identification system for Enterobacteriaceae (Micronaut‐E, Merlin‐Diagnostika, Bornheim, Germany). Natural resistance was found to benzylpenicillin, oxacillin, the macrolides except azithromycin, lincosamides, streptogramins, glycopeptides, rifampicin and fusidic acid. If you do not receive an email within 10 minutes, your email address may not be registered,

When the natural population was clinically resistant, it was described as naturally (intrinsically) resistant. The method has been described in detail previously [There were no, or only small, species‐related differences in natural susceptibility to most antimicrobial agents.












While 51 of 213 (23.9%) Enterobacter cloacae isolates were colistin-resistant, only six of 143 (4.2%) E. a …

Antibiotic resistance occurs when the germs no longer respond to the antibiotics designed to kill them.

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Universität Bonn, Institut für Medizinische Mikrobiologie und Immunologie, Pharmazeutische Mikrobiologie, Meckenheimer Allee 168, D‐53115 Bonn, GermanyUniversität Bonn, Institut für Medizinische Mikrobiologie und Immunologie, Pharmazeutische Mikrobiologie, Meckenheimer Allee 168, D‐53115 Bonn, GermanyUniversität Bonn, Institut für Medizinische Mikrobiologie und Immunologie, Pharmazeutische Mikrobiologie, Meckenheimer Allee 168, D‐53115 Bonn, GermanyUniversität Bonn, Institut für Medizinische Mikrobiologie und Immunologie, Pharmazeutische Mikrobiologie, Meckenheimer Allee 168, D‐53115 Bonn, GermanyUse the link below to share a full-text version of this article with your friends and colleagues. They are resistant because they produce an enzyme called a carbapenemase that disables the drug molecule.